A delicious dry cocktail with prominent notes of apricot. I’ve been featuring an awful lot of heavy, brown liquor drinks lately. That’s fine – I happen to love brown liquor! But I also love gin, and particularly love gin drinks that are crystal-clear, crisp, dry, and light on the palate, whether it be the classic Martini, the delicious H&H, or my own Beryl. So when I ran across this interesting Martini variant in Julie Reiner’s terrific book, The Craft Cocktail Party, I knew I had to give it a shot. Reiner is responsible for both the first bar at which I ever tried a craft cocktail (The Flatiron Lounge) and my favorite bar in Brooklyn (Clover Club), and she’s a celebrated professional who’s respected throughout the world. She knows what she’s doing! The use of apricot eau-de-vie is really interesting, here. As I talked about in our Gentleman’s Introduction to Brandy, eaux-de-vie are not sweet. They’re basically vodka that’s distilled from fruit and thus retains some of the fruit’s natural scent and flavor. This means the Gin Blossom is still a pretty dry drink, like a Martini, but with a substantial hit of apricot, especially in the nose. If you haven’t ever picked up an eau-de-vie before, I strongly recommend it. They’re fantastic as secondary ingredients for cocktails, and can really add to a drink without making it overly sweet or cloying. Items in parentheses are what I used when making this drink, and are included as recommendations.
 A delicious dry cocktail with prominent notes of apricot.
Let us know how it was!
Gin blossoms are an allergic reaction to alcohol that causes the face and neck--and sometimes other areas of the body such as the torso--to become red and warm. This condition is known as alcohol flush reaction or Asian flush reaction because of its high prevalence in the Asian population. Alcohol flush reaction has been known in the Asian populations for thousands of years, but no one knew what caused it until recently. In 2010, a researcher named Bing Su discovered that alcohol flush reaction has a genetic cause. The allele ADH1B*47 has a mutation that causes alcohol flush reaction, and that mutation developed around the same time as rice was first domesticated and fermented into alcohol. Bing Su theorizes that this reaction is an evolutionary development that has a protective effect against alcoholism, which is borne out by the fact that individuals with alcohol flush reaction have a lower risk of alcoholism.
CausesAlcohol flush reaction is caused by an allergic reaction to alcohol 1. When you consume alcohol, your body starts a complex process to break it down. First, the alcohol is broken down into acetaldehyde by an enzyme called alcohol dehydrogenase, or ADH. Once that happens, a second enzyme called ADH2 breaks the acetaldehyde into acetic acid, and after that other enzymes break the acetic acid into carbon and water. The deficiency in people with alcohol flush reaction occurs with the ADH2 1. Instead of producing ADH2, a defective enzyme is produced. As a result, instead of being broken down, the acetaldehyde enters the bloodstream, where it causes the characteristic flush reaction.
Alcohol flush reaction is not a harmless flush 1. Acetaldehyde, the product of alcohol that is not broken down by the defective enzyme, is a toxic substance. In fact, acetaldehyde is even more toxic than alcohol and is the substance that causes a hangover. For those with alcohol flush reaction, facial flushing, sleepiness, increased heart rate, low blood pressure and dizziness can result from the buildup of acetaldehyde in the system, making them feel very ill even after a small amount of alcohol. Over time, the consumption of alcohol can lead to organ damage and even death.
TreatmentThe best treatment for alcohol flush reaction is not to drink or to cease drinking once symptoms of the flush begin. Some sufferers advocate consuming starchy or sugary foods before drinking because the starch and sugar coat the stomach and keep the symptoms at bay. Others recommend taking products that contain Famotidine, an ingredient found in heartburn relief medications. These remedies no not prevent alcohol flush reaction, as they do not compensate for the defective enzyme, so the best solution remains to abstain from drinking.
Rhinophyma is a condition causing development of a large, bulbous nose associated with granulomatous infiltration, commonly due to untreated rosacea.[1] The condition is most common in older white males.[2] Colloquial terms for the rhinophyma include "whiskey nose", "gin blossom", "toros nose", and "potato nose".[2] Rhinophyma is characterised by prominent pores and a fibrous thickening of the nose, sometimes with papules.[3] It is associated with the common skin condition rosacea and it can be classified clinically into 5 grades of increasing severity.[4] ComplicationsTissue thickening may come to cause airway obstruction and impede breathing.[2] Rhynophyma cause psychological distress due to its effect on one's personal appearance[5][2] and social perceptions of a link with alcoholism.[2] Rhinophyma develops in certain individuals with an unknown predisposition from long-standing rosacea which has progressed to a severe form.[2] Although rhinophyma has been commonly presumed to be linked to alcoholism, a direct causal relationship between the condition and excessive alcohol consumption has not been substantiated.[2] Alcohol may cause increased flushing in those affected.[6] Rhinophyma develops in some individuals after long-standing rosacea that has progressed to acne rosacea.[2] Rosacea usually commences in people between the age of 20-30 years. Rosacea begins with facial flushing (pre-rosacea). The nasal skin then thickens and hypervascularises, leading to persistent erythema (vascular rosacea). Papules and pustules then develop, marking the beginning of acne (inflammatory) rosacea. A subset of those affected by acne rosacea go on to develop rhinophyma. Chronic infection is common as the thickened sebaceous fluid traps bacteria.[2] Rhinophyma is characterised by hypertrophy of nasal skin, with hyperplasia and fibrosis of the sebaceous glands and connective tissue. The nasal tip and alae are preferentially affected by the hypertrophy, and the lower portion of the nose is predominantly affected. The underlying bony structure is not affected.[2] The exact pathophysiological mechanism underlying the development of rosacea and rhinophyma is unknown, but a combination of neurovascular and immune disturbance is thought to be involved, causing inflammation, fibrosis, and vascularisation.[2] Male predispositionEven though females are more often affected by acne rosacea, they seldom go on to develop rhinophyma. It is thought that androgenic factors predispose men to develop rhynophyma.[2] Rhinophyma is diagnosed clinically based on presentation (erythema, telangiectasias, and nasal skin hypertrophy). Diagnosis is confirmed by histology.[2] Treatment consists of paring down the bulk of the tissue with a sharp instrument or carbon dioxide laser and allowing the area to re-epithelialise. Sometimes, the tissue is completely excised and the raw area skin-grafted.[7] Rhynophyma is most common in males of European heritage over the age of 50. Males are 5 to 30 times as likely to be affected as females, possibly due to androgenic factors.[2] In the United States, people of predominantly Asian and African ancestry are only rarely affected by the condition.[2] Colloquial names for the condition include "whiskey nose", "gin blossom", and "potato nose". The condition has often been presumed to be a result of alcoholism (even though it remains unclear whether alcohol actually contributes to the development of rhynophyma) which has led to stigmatisation of people with the condition. In film, villainous characters have been portrayed as having rhynophyma, notably the evil queen in the animated film Snow White and the Seven Dwarfs.[2] Among those people known to have had the disease was the American banker J. P. Morgan.[8]
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What is a gin blossom
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